Type 2 Diabetes (T2D) has become a major public concern and based on WHO reports, it is considered to be one of the most commonly diagnosed chronic conditions worldwide. Senescence is one of the hallmarks of diabetes. Endocrine disruptors like bisphenol A (BPA), are set of chemicals that mimic endocrine hormones and are known to interfere with endocrine functions. In our current study, we used zebrafish embryo and larvae to study the effects of bisphenol A exposure on senescence and its impact during metabolic stress. Zebrafish embryos were exposed to a range of BPA concentration form 3 to 6 days post fertilization (dpf) and fixed at 6 dpf. Gene expression analysis of senescence markers -β-gal, p21, p53, p63 are done by qPCR. Senescence associated β-galactosidase assay was carried out for the confirmatory purpose. Telomerase activity assay and telomere restriction fragment analysis were done to analyse the effects of BPA on telomere shortening directly. In addition, rate of glucose oxidation in the presence of bisphenol A was analysed using the Seahorse extracellular flux analyser. Altogether our results indicates the impact of environmental contaminant, bisphenol A on accelerated aging in age associated metabolic disease like diabetes.